Education and Training: Post-doctoral Fellowship: University of Texas Southwestern Medical Center at Dallas and Presbyterian Hospital of Dallas; Ph.D., University of Utah; M.S. and B.S., University of Wyoming
Additional Technique Training: Autonomic Neuroscience - Beth Israel Deaconess Medical Center, 2010; Integrative Physiology - Rigshospitalet/University of Copenhagen, 2008 & 2004; Autonomic & Cardiovascular Physiology - Penn State College of Medicine, 2003.
Research Interests and Current Projects:
Medical Applications of Environmental Physiology
Humans do not live in isolation from either their natural or built environments. Ambient conditions cause reflex physiological responses, which can be extensive in extreme environments (such as an uncompensable heat stress) and are often different in healthy people compared to those with disease. Both warm and cold ambient conditions are associated with increased morbidity and mortality; in addition, as the climate warms, there will be additional factors such as urban heat island effects and more extreme weather patterns. Some example interactions of health, disease, and the thermal environment we study include:
- Worker health and safety
- Hyperhidrosis and eccrine sweat gland function
- Rosacea and facial flushing
- Heat syncope and cryotherapy
Metzler-Wilson, K., Toma, K., Sammons, D.L., Mann, S., Jurovcik, A. J., Demidova, O., & Wilson, T. E. Augmented supraorbital skin sympathetic nerve activity responses to symptom trigger events in rosacea patients. Journal of Nuerophysiology, in press (DOI: 10.1152/jn.00458.2015).
Gray, B. D., Metzler-Wilson, K., Dawes, K. W., & Wilson, T. E. A neural link to understanding rosacea: Focusing on flushing triggers. Journal of the American Osteopathic College of Dermatology, in press.
Wilson, T. E. & Metzler-Wilson, K. Sweating chloride bullets: Understanding the role of calcium in eccrine sweat glands and possible implications for hyperhidrosis. Experimental Dermatology, 24: 177-178, 2015.
Crandall, C. G. & Wilson, T. E. Human cardiovascular responses to passive heat stress. Comprehensive Physiology, 5: 17-43, 2015.